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Adjunctive therapies in idiopathic pulmonary fibrosis—where do we stand?

   
@article{JTD25904,
	author = {Brandon Nokes and Kamyar Afshar},
	title = {Adjunctive therapies in idiopathic pulmonary fibrosis—where do we stand?},
	journal = {Journal of Thoracic Disease},
	volume = {11},
	number = {2},
	year = {2018},
	keywords = {},
	abstract = {IPF is a restrictive lung disease that is characterized by dyspnea, non-productive cough and progressive loss of lung function. Some patients with IPF will have a slow and steady decline in their lung function, while others can experience a more rapid deterioration in their health and documented lung functions (1). The pathogenesis of IPF [histologically referred to as usual interstitial pneumonia (UIP)], is incompletely understood. However, it may involve a number of genes including surfactant protein C and telomerase mutations, resultant pneumocyte senescence, and aberrant repair pathways hallmarked by progressive pulmonary fibrosis (2,3).},
	issn = {2077-6624},	url = {https://jtd.amegroups.org/article/view/25904}
}